No matter if you live in an apartment or house, or you just would like to keep your home free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to restrict the use of electronic devices. You can also turn to EMF block paint to stop EMF radiation from entering your home. Another method to protect your house against EMF radiation is to put up a RF shielding canopy. It is a type made of net which contains EMF shielding. It's used to block EMFs from entering a space. Another alternative is to have your home equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
A number of studies have proven how the EMF that is not ionized produces antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This may account for the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on cancer patients isn't evident.
Effects on the effects of AM RF EMFs on HCC tumour growth in vivo were examined in mice. emf blocking were divided into three groups. emf blocker was not exposed to RF EMF. Another group of participants was subjected to RF EMF at the same frequency to that of humans. The third group was exposed to RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumors was evaluated against that of RCF. The results showed that the tumors treated by HCCMF had significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism of tumor-specific AM RF EMF may be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is controlled through CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H may have broader implications for the diagnosis and treatment of a variety of cancers.
The tumors in those in the group that were unaffected to RF EMF, and were fed a standard mouse diet. The tumours in the HCCMF group were infected with Huh7 cells after they were between five and seven weeks old. The tumours were then euthanized when they showed excessive burden.
The tumors of the three groups also showed different growth curves. The HCCMF-treated tumors saw a significant decrease in the size of the tumour after 8 weeks. However, the tumors which were treated by RCF didn't show shrinkage. The difference was highly significant. The tumors treated by RCF were able to show necrosis, which is common in tumors that have been that are exposed to RCF. The possibility is that the necrosis is caused by the lack of oxygen in the more invasive cancers.
In sum, the results indicate an AM-RF EMF is a powerful source of anti-cancer properties in vitro as well as in vivo. emf blocking have shown that AM RF EMF produces measurable shrinkage of tumors for HCC patients. There is a possibility that AM RF EMF triggers these effects due to CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting impact on the development of HCC tumors in vivo.