No matter if you live in an apartment or a house or simply would like to keep your home free from EMFs There are a variety of methods to reduce exposure. One of the most straightforward is to restrict the use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from entering your home. Another way to shield your house from EMF radiations is to install a RF shielding canopy. This is a cloth made of net which contains EMF shielding. It is utilized to stop EMFs from entering a room. Another option is to get your home fitted with a conductive enclosure. These devices are called Faraday cages.
A number of studies have proven studies have shown that the nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation in cancer-related stem cells. This could be the reason for the long-term response seen in patients with advanced HCC. However, the reason behind AM EMF's effects on patients with cancer is not clear.
Effects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumors were divided in three different groups. The first group was not exposed to RF EMF. Second group members were exposed RF EMF at the same frequency to that of humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF had significant shrinkage. However, emf blocker treated with RCF didn't show evidence of tumour shrinkage.
The mechanism of tumour-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is controlled by CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications for diagnosis and treatment of various cancers.
emf blocker in the control group were not exposed to EMF from radiofrequency, and fed a normal mouse diet. The tumours in the HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumours were then euthanized after they had a high burden.
The tumours in the three groups also showed different growth curves. The tumors treated with HCCMF saw a significant decrease in the size of the tumour after eight weeks. However, emf blocking that were treated using RCF showed no reduction in size. The difference was highly significant. The tumors treated by RCF were able to show necrosis, which is common in tumours exposed to RCF. The possibility is that this necrosis was due to an absence of oxygen in larger cancers.
In conclusion, the findings suggest that AM RF EMF exhibits anticancer activity in vitro and in the vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors in HCC patients. The possibility is that the AM EMF produces these effects because of CACNA1H, a protein involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the development of HCC tumours in vivo.