Whether you live in an apartment or a house or just need to ensure that your house is free from EMFs, there are a number of ways you can reduce exposure. One of the most effective is to restrict the use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another method to protect your home against EMF radiation is to put up an RF shielding canopy. This is a cloth of net that has EMF shielding. It's used to prevent EMFs from entering a room. Another option is to get your house equipped with an enclosure that is conductive. emf blocker are referred to as Faraday cages.
Numerous studies have demonstrated how the EMF that is not ionized has anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This could explain the long-term response seen in patients with advanced HCC. But, emf blocking for AM EMF's impact on cancer patients isn't evident.
Effects of AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumours were divided into 3 groups. One group did not have exposure to RF EMF. Second group members were exposed to RF EMF at the same frequency to that of humans. In the third, they were subjected the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumors was compared to that of RCF. The results revealed that tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of shrinkage of the tumor.

The mechanism behind tumour-specific AM RF EMF could be based on the fact that cancer cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is controlled by CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H may have broader implications in the diagnosis and treatment of a variety of cancers.
The tumors in the controls were never exposed EMF from radiofrequency, and fed a standard mouse diet. The tumors of HCCMF HCCMF group were injected with Huh7 cells after they were five-seven weeks old. The tumours were then euthanized after they had a high burden.
The tumours in the three groups also showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after eight weeks. However, emf blocking treated with RCF didn't show shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis that is common in tumors that have been exposed to RCF. The possibility is that the necrosis was due to the lack of oxygen in larger tumours.
In sum, the results indicate that AM RF EMF is a powerful source of anti-cancer properties in vitro as well as in live. A number of studies have proven it is true that AM RF EMF produces measurable tumour shrinkage in HCC patients. It is possible that AM RF EMF triggers these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting impact on the development of HCC tumours in living tissue.