If you reside in an apartment or house or just need to ensure that your house is free from EMFs There are plenty of ways you can reduce exposure. One of the most effective is to reduce the usage of your electronic devices. https://telegra.ph/EMF-Block-Paint-and-even-EMF-Shielding-04-08-17 can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another easy way to protect your house against EMF radiation would be to use a RF shielding canopy. It is a type made of net which contains EMF shielding and is used to prevent EMFs from entering a space. Another alternative is to have your home equipped with an electrical enclosure. They are referred to as Faraday cages.
Several studies have shown that the non-ionizing RF EMF can cause anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed result from the deregulation of cancer stem cells. emf blocker could explain the long-term responses seen in some patients with advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not yet clear.
Effects from AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumours were classified in three different groups. The first group was not exposed RF EMF. Second group members were exposed to RF EMF at a frequency that is similar to that used in humans. The third group was exposed to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumors treated by HCCMF showed significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism behind tumor-specific AM RF EMF could be driven by the fact that cancer cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is controlled by CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H may have broader implications in the diagnosis and treatment of various cancers.
The tumours in the controls were never exposed EMF from radiofrequency, and fed a normal mouse diet. The tumors of HCCMF HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed when they showed excessive burden.
The tumors of the three groups showed distinct growth curves. The tumors treated with HCCMF saw a significant decrease in tumour size after 8 weeks. However, tumors treated with RCF did not show any shrinkage. The difference was highly significant. The tumors treated by RCF were able to show necrosis that is common in tumours that are exposed to RCF. There is a possibility that this necrosis was caused by a lack of oxygen in the more invasive tumours.
In summary, the results show the fact that AM EMF has anti-cancer properties in vitro as well as in vivo. emf blocker have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage for HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained impact on the development of HCC tumors in living tissue.