Whether you live in an apartment or house or just need to ensure that your house is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the most effective is to limit your electronic device use. It is also possible to use EMF blocker paint to block EMF radiation from entering your house. Another way to shield your house from EMF radiation is to use an RF shielding cover. It is a type of net that contains EMF shielding. It is utilized to block EMFs from entering a space. Another option is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF can cause antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to involve down-regulation the cancer stem cell. This may account for the long-term response seen in patients with advanced HCC. But, the reason for AM EMF's effects on patients with cancer is not yet clear.
Effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumors were classified in three different groups. emf blocker was not exposed RF EMF. Another group of participants was subjected to RF EMF at a frequency that is similar to that used in humans. In the third, they were subjected to the RF EMF in HCC-specific frequencies. The effect of HCCMF on the tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF showed significant shrinkage. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.
The reason for cancer-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is caused by CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have more broader implications for the diagnosis and treatment of a variety of cancers.
The tumours of those in the group that were unaffected to EMF from RF, and fed a normal mouse diet. The tumours in HCCMF HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed after they had a high burden.
The tumours in the three groups also showed distinct growth curves. The tumours treated with HCCMF saw a significant decrease in the size of the tumour after eight weeks. However, the tumors that were treated using RCF did not show any reduction in size. The difference was highly significant. emf blocking treated by RCF had necrosis, which is common in tumours that are exposed to RCF. It is possible that the necrosis is caused by the lack of oxygen in larger cancers.
In summary, the results show that AM RF EMF is a powerful source of anticancer activity in vitro and in the vivo. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is emf blockers that AM RF EMF produces these effects because of CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the growth of HCC tumours in vivo.