EMF Blocking and even EMF Shielding

· 2 min read
EMF Blocking and even EMF Shielding

If you reside in an apartment or a house or simply want to keep your home free of electromagnetic fields There are a variety of methods to reduce exposure.  emf blockers  of the most effective is to restrict your use of electronic devices. You could also consider EMF blocker paint to block EMF radiation from reaching your house. Another easy way to protect your house from EMF radiations is to use a shielding canopy for RF. This is a cloth made of net which contains EMF shielding and is used to prevent EMFs from entering a room. Another option is to have your house equipped with an enclosure that is conductive. These devices are called Faraday cages.

Numerous studies have demonstrated studies have shown that nonionizing EMF has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation the cancer stem cell. This may account for the long-term response seen in patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not clear.

click here  on the effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumours were divided into 3 groups. The first group was not exposed to RF EMF. The second group was exposed RF EMF at the same frequency to that used in humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results showed that the tumours treated with HCCMF were significantly shrinking. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.

The mechanism behind tumour-specific AM RF EMF might be based on the fact that cancer cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is controlled through CACNA1H the protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications for treatment and diagnosis of many cancers.

emf blocker  in the controls were never exposed RF EMF, and were fed a standard mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized when they showed excessive burden.

The tumours in the three groups showed distinct growth curves. The HCCMF-treated tumors saw a significant reduction in tumour size after 8 weeks. However, tumors which were treated by RCF didn't show shrinkage. The difference was highly significant. The tumors treated by RCF were able to show necrosis that is common in tumors that have been that are exposed to RCF. The possibility is that the necrosis was due to an absence of oxygen in the more invasive cancers.



In sum, the results indicate the fact that AM EMF is a powerful source of anticancer activity in vitro and in the vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. The possibility is that the AM EMF produces these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting effect on the growth of HCC tumors in vivo.