If you reside in an apartment or a house or simply would like to ensure your home is free of electromagnetic fields There are a variety of methods to reduce exposure. emf blocker of the most effective is to limit the use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your house. Another method to protect your home from EMF radiation would be to install a RF shielding canopy. This is a fabric made of net which contains EMF shielding. It is utilized to block EMFs from entering rooms. emf blocking is to have your house equipped with an enclosure that is conductive. These devices are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed be based on the downregulation the cancer stem cell. This could explain the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not yet clear.
Aspects from AM RF EMFs on HCC tumor growth in vivo were studied in mice. The tumors were split into 3 groups. The first group was not exposed to RF EMF. The second group was exposed to RF EMF at a frequency that is similar to the one used by humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on the tumours was evaluated against that of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism of tumor-specific AM RF EMF may be based on the fact that tumor cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is caused through CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.
The tumours in the controls were never exposed to RF EMF, and were fed a normal diet of mice. The tumours in the HCCMF group were treated with Huh7 cells after they were 5 to 7 weeks old. block emf were then euthanized in cases of excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF saw a significant reduction in the size of the tumour after 8 weeks. However, the tumours which were treated by RCF showed no signs of shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis that is common when tumors are exposed to RCF. It is possible that the necrosis is caused by the lack of oxygen in the larger tumours.
In summary, the results suggest that AM RF EMF has anticancer properties in vitro as well as in live. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors in HCC patients. The possibility is that the AM EMF produces these effects through CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained influence on the growth of HCC tumors in vivo.