https://brandyotter17.bravejournal.net/post/2023/04/08/EMF-Block-Paint-and-even-EMF-Shielding4 if you live in an apartment or a house, or you just want to keep your home free of EMFs, there are a number of ways you can reduce exposure. One of the most effective is to limit your use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from reaching your home. Another way to shield your home against EMF radiation is to put up an RF shielding canopy. Helpful resources is a type of net that contains EMF shielding. It is utilized to block EMFs from entering a space. Another alternative is to have your house equipped with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF produces antiproliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation in cancer-related stem cells. This may account for the long-term response seen in some patients with advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not evident.
The effects on the effects of AM RF EMF on HCC tumor growth in vivo was studied in mice. The tumours were divided in three different groups. One group did not have exposure to RF EMF. Another group of participants was subjected to RF EMF at a frequency that is similar to the frequency used by humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumours was evaluated against that of RCF. The results showed that the tumours treated with HCCMF were significantly shrinking. However, the tumors treated with RCF did not show any evidence of tumour shrinkage.
The mechanism behind cancer-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is controlled through CACNA1H which is a protein that is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.
https://lipcrate37.werite.net/post/2023/04/08/EMF-Blocking-and-EMF-Shielding2 of those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumours in HCCMF HCCMF group were infected with Huh7 cells at the time they were five-seven weeks old. The tumors were then killed after they had a high burden.
The tumours from the three groups showed distinct growth curves. The tumours treated with HCCMF saw a significant reduction in tumour size after 8 weeks. However, the tumors which were treated by RCF didn't show shrinkage. The difference was significant. The tumours treated with RCF were able to show necrosis that is common when tumors are exposed to RCF. There is a possibility that this necrosis was caused by an absence of oxygen in larger tumors.
In sum, the results show the fact that AM EMF exhibits anticancer activity in vitro and in vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF causes these effects due to CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained influence on the growth of HCC tumors in living tissue.